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Pathways of neurodegeneration - multiple diseases
Core of basic research: Decipher common pathogenic mechanisms across multiple diseases, including protein misfolding and aggregation, oxidative stress, autophagic dysfunction, neuroinflammation, and abnormal apoptotic pathways, exploring the crosstalk between these pathways and potential common intervention targets.
Core key proteins: α-Syn/tau/mHTT/SOD1 (disease-related proteins with misfolding and aggregation), LC3/p62/Beclin-1 (autophagy-related proteins with dysfunction), TNF-α/IL-6 (neuroinflammatory factors), Caspase family (apoptosis-executing enzymes), Bcl-2 (anti-apoptotic protein).
Core key proteins: α-Syn/tau/mHTT/SOD1 (disease-related proteins with misfolding and aggregation), LC3/p62/Beclin-1 (autophagy-related proteins with dysfunction), TNF-α/IL-6 (neuroinflammatory factors), Caspase family (apoptosis-executing enzymes), Bcl-2 (anti-apoptotic protein).
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