Gastric cancer
Core of basic research: Focus on Helicobacter pylori (Hp) infection-induced inflammatory microenvironment, epithelial-mesenchymal transition (EMT) mechanisms, and abnormal activation of receptor tyrosine kinases such as EGFR and c-Met. Explore the association between CDH1 (E-cadherin) inactivation-induced cell adhesion disorders and invasion/metastasis.
Core key proteins: EGFR/HER2 (receptor tyrosine kinases activating proliferation signals), c-Met (hepatocyte growth factor receptor promoting invasion), PI3K/Akt (core of signaling pathway), TP53 (tumor suppressor gene with frequent mutations), CDH1 (E-cadherin, loss of adhesion upon inactivation), RUNX3 (transcription factor regulating differentiation and apoptosis), VEGF (angiogenic factor promoting blood supply).
Core key proteins: EGFR/HER2 (receptor tyrosine kinases activating proliferation signals), c-Met (hepatocyte growth factor receptor promoting invasion), PI3K/Akt (core of signaling pathway), TP53 (tumor suppressor gene with frequent mutations), CDH1 (E-cadherin, loss of adhesion upon inactivation), RUNX3 (transcription factor regulating differentiation and apoptosis), VEGF (angiogenic factor promoting blood supply).
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